This is a great supplement I have added a lot of information about it you can get it at

http://synergymuscle.com/catolalog/index.php


it is called cAMPHIBOLIC

What it does?

--Liberates fat from fat cells like Clen and Ephedrine. Unlike the others, cAMPHIBOLIC by passes the beta receptors so downregulation of receptors is not an issue. Clen and Ephedrine lose it's effects after 2-4 weeks plus clen can damage the heart and with ephedrine some get anxiety, jitters and crash after 2 hours later which is almost an addictive drug feel. With cAMPHIBOLIC, You just feel HOT and with a nice sustained energy.

cAMPHIBOLIC acts independently of beta receptors, even cells whose beta receptors have been severely downregulated by exposure to beta2 agonists are still responsive to forskolin. So a person who has been using clenbuterol or albuterol over an extended period might consider switching to a cAMPHIBOLIC, achieving the same net result of fat loss by bypassing the downregulated beta-receptors.

--Boost thyroid levels so you burn fat while resting!! You can liberate all the fat but with out elevating metabolic rate fat will redeposit in fat cells. cAMPHIBOLIC liberates fat and burns fat at REST. forskolin promotes the conversion of the relatively inactive T4 to its potent metabolite T3.

--Increace Mood--several studies have shown that patients suffering from depression have reduced levels of activity of cAMP( cyclic adenosine monophosphate ) signaling in the brain. cAMPHIBOLIC increase cAMP!

--Increase Sexual desire--It also has the ability to relax smooth muscles in the penis, allowing the organ to fill with blood. If you really want to punish the misses add cAMPHIBOLIC with Viagra and cialis. synergistic effect on erection when forskolin was combined with Viagra. So the agents alone promote erection, and when combined exhibit synergism, an effect greater than the sum of the two!

--Increase Testosterone production--Testosterone is produced in testicular Leydig cells when LH binds to surface receptors and initiates a signaling cascade involving elevated levels of cAMP. This cascade leads ultimately to increased levels of an important Leydig cell cholesterol transfer protein (16) (testosterone is made from cholesterol) and activation of steroidogenic enzymes involved in testosterone production (17). So by elevating levels of cAMP, (with forskolin) an intermediate in the signaling cascade that ranges from LH binding to testosterone production, we should see an increase in testosterone output...

FOR THE OLDER MEN ON THE FORUM!
This may be particularly important for those of us who are feeling the effects of aging. testosterone production declined with age as a function of declining cAMP levels. So something is keeping LH from elevating cAMP and inducing steroidogenesis.

--elevating cAMP promotes anabolism--AAS users are all too familiar with the latter effect, when hard-earned gains rapidly disappear post-cycle. Much of this atrophy is caused by the action of the so-called calpains. The calpains are a family of calcium dependent enzymes that degrade unused muscle tissue. Calpains in turn are inhibited by another endogenous compound called calpastatin. How is this related to forskolin? It turns out that calpastatin is upregulated by cAMP . Thus not only does cAMPHIBOLIC promote anabolism, it slows muscle catabolism when muscle is not being used extensively. This would be expected to be important when a person is immobilized or unable to train due to injury, or is simply unable to maintain the rigorous training regimen off cycle that they adhered to while on AAS.

--REDUCE CORTISOL--antiglucocorticoid properties of cAMPHIBOLIC are likely of much more interest to the average bodybuilder or athlete, since endogenous glucocorticoids, particularly cortisol, are not only catabolic to bone, but catabolize muscle tissue as well. By exerting an anabolic, antiglucorticoid effect cAMPHIBOLIC helps preserve muscle tissue during times of physical and emotional stress.

--Stop GLUCOSE UPTAKE IN fat cells--this appears to be a cAMP independent phenomenon, with cAMPHIBOLIC directly inhibiting the glucose transport. This is likely another mechanism whereby glucose promotes fat burning. With limited glucose for use as fuel, fat cells must rely on their own stored fat to carry out the metabolic processes required for their survival.

http://www.synergymuscle.com/catalog/



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