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Old Sun, Dec-13-09, 11:48
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Hutchinson Hutchinson is offline
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Plan: Dr Dahlqvist's
Stats: 205/152/160 Male 69
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Progress: 118%
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Perhaps as this thread is about Fructose and the dangers thereof it may be worth getting back to the topic.

Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans

they took at group of 39 overweight and obese people who consumed glucose- or fructose-sweetened beverages providing 25% of energy requirements for 10 weeks. It was a double-blinded parallel arm study that used matched subjects. So no one knew who was on glucose or fructose beverages.

The increase in Visceral adipose tissue in subjects consuming fructose and the increase in the expression of lipogenic genes in Subcutaneous adipose tissue in subjects consuming glucose suggest that fructose and glucose have differential effects on regional adipose distribution.

In addition increases of postprandial triglyceride and fasting and postprandial apoB, they show that fructose consumption increases plasma concentrations of fasting sdLDL, oxidized LDL, and postprandial RLP-C and RLP-TG in older, overweight/obese men and women, but glucose consumption does not.

Fructose consumption increased hepatic fractional de novo lipogenesis (DNL), and postprandial lipoprotein lipase (LPL) activity was lower in subjects consuming fructose compared with those consuming glucose. These results suggest that both increased DNL and decreased LPL-mediated clearance contribute to fructose-induced postprandial hypertriglyceridemia.

Consumption of fructose at 25% of energy requirements with an ad libitum diet decreased glucose tolerance and insulin sensitivity in older overweight/obese adults compared with glucose consumption.
(this is clear evidence HFCS causes Diabetes type 2)

Visceral Adipose Tissue (fat round waist) accumulation and increases of 24-hour triglyceride exposure, peak postprandial Triglyceride concentrations, and postprandial remnant-like particle lipoprotein–TG concentrations in response to fructose consumption were more pronounced in men than in women.

Consumption of sugar-sweetened beverages resulted in greater decreases in insulin sensitivity in women than in men.
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