I make my own, found a good recipe on line. I reduce the tomatoes and vinegar, in effect doubling the spices. Everyone likes it.
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Perhaps as this thread is about Fructose and the dangers thereof it may be worth getting back to the topic.
Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans they took at group of 39 overweight and obese people who consumed glucose- or fructose-sweetened beverages providing 25% of energy requirements for 10 weeks. It was a double-blinded parallel arm study that used matched subjects. So no one knew who was on glucose or fructose beverages. The increase in Visceral adipose tissue in subjects consuming fructose and the increase in the expression of lipogenic genes in Subcutaneous adipose tissue in subjects consuming glucose suggest that fructose and glucose have differential effects on regional adipose distribution. In addition increases of postprandial triglyceride and fasting and postprandial apoB, they show that fructose consumption increases plasma concentrations of fasting sdLDL, oxidized LDL, and postprandial RLP-C and RLP-TG in older, overweight/obese men and women, but glucose consumption does not. Fructose consumption increased hepatic fractional de novo lipogenesis (DNL), and postprandial lipoprotein lipase (LPL) activity was lower in subjects consuming fructose compared with those consuming glucose. These results suggest that both increased DNL and decreased LPL-mediated clearance contribute to fructose-induced postprandial hypertriglyceridemia. Consumption of fructose at 25% of energy requirements with an ad libitum diet decreased glucose tolerance and insulin sensitivity in older overweight/obese adults compared with glucose consumption. (this is clear evidence HFCS causes Diabetes type 2) Visceral Adipose Tissue (fat round waist) accumulation and increases of 24-hour triglyceride exposure, peak postprandial Triglyceride concentrations, and postprandial remnant-like particle lipoprotein–TG concentrations in response to fructose consumption were more pronounced in men than in women. Consumption of sugar-sweetened beverages resulted in greater decreases in insulin sensitivity in women than in men. |
It's important to keep in mind that sucrose (aka sugar) is 50% fructose. Only slightly less nasty than HFCS.
Although we also heard recently that a lot of HFCS is also contaminated with mecury. Whee! |
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I've seen it in the supermarket within the last few months. If they have indeed stopped making it it would have to have been very recently. |
Are You Pouring on the Pounds?
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Fructose ingestion acutely elevates blood pressure in healthy young humansSome interesting graphs here showing the effect of fructose on blood pressure. Quote:
I think hydroxymethylfurfural is probably a bigger threat. |
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I suddenly feel sick. |
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Thanks for the link/info! I will have to go back and read it more closely. On a fast skim through it reads like a technical manual. |
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In this case we see Stanhope was responsible for this paper Fructose consumption: considerations for future research on its effects on adipose distribution, lipid metabolism, and insulin sensitivity in humans. Quote:
I find taking it sentence by sentence helps me grasp some of what they are talking about, but going back to some of their earlier work helps to see where they are coming from and that helps a create a better understanding their most recent findings. |
Great find. That study pretty much isolates fructose as the cause of metabolic syndrome, at least the more dangerous components. This also puts to bed the claim that fructose is "better for diabetics" because it's supposedly less insulinogenic.
Kudos to the study sponsors for making it open access as well. |
Yeah, it's all about fructose. Really bad stuff when refined. At least whole fruits comes with some antidotes to it making them almost ok.
Patrick |
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